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Iron overload enhances oxidative stress and the NACHT, LRR, and PYD domains-containing protein 3 (NLRP3) inflammasome signaling, triggering the evolution of several inflammatory mediators, resulting in a cascade of inflammatory responses and renal dysfunction in iron-overloaded rats (Chaudhary et al., 2018)

Atorvastatin increases LEENE expression, thereby upregulating eNOS expression, while inhibition of LEENE strongly enhances ECs-monocyte adhesion induced by pulsatile shear stress (Miao et al., 2018)

however, the underlying mechanisms of its antitumoral function in relation to cancer cells require further exploration (Fig

The therapeutic implications of ketone bodies: the effects of ketone bodies in pathological conditions: ketosis, ketogenic diet, redox states, insulin resistance, and mitochondrial metabolism

EA at LI11 and ST36 reduced infarct volume by attenuating the overactivation of Iba-1 and ED1-positive microglia in the peri-infract sensorimotor cortex, decreasing the level of TNF-, IL-1, and IL-6 in serum, and preventing the nucleus translocation of NF-B p65 and the expression of p38 mitogen-activated protein kinase (p38 MAPK) and myeloid differentiation primary response gene 88 (MyD88) in the peri-infract sensorimotor cortex