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Figure 6 Intracellular GSH Is a Regulator of BDG-Induced NO Secretion, Phagocytosis, and Cytokine Production As metabolism of myeloid cells is closely linked to their effector functions and intracellular GSH levels regulated BDG-induced metabolic changes, we wanted to investigate if GSH could also regulate monocytes effector functions in response to BDG

buffer B acetonitrile, 0.1% TFA) with a C18 Hypersil-ODS (5 m, 250 4.6 mm) column and a flow rate of 1 ml/min

However, the absence of cysteine leads to insufficient GPX4 synthesis, increased expression of wild-type p53, and increased cell sensitivity to ferroptosis (Bretscher et al., 2015)

The analogues, 1,8-diMQ and 5-amino-1MQ were tested at concentrations from 200 M-10 nM and 600 M-10 nM, respectively