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The precise mechanism may involve multiple processes including compensatory receptor crosstalk and it is to be further investigated

La sostanza non e stata testata, approvata o commercializzata come prodotto medicinale finito

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In cases of abnormal cellular iron metabolism, the excess accumulation of iron prompts intracellular accumulation of reactive oxygen species and lipid peroxidation, resulting in the degradation of cellular constituents such as lipids, DNA, and proteins (Koleini et al., 2021) (Figure 1)

The combination of enhanced endogenous antioxidant gene repertoires (i.e., expansion of PRDX family) and a marked abundance of n-3 PUFA cardiac phospholipids enhances the overall resistance of whales toward cardiovascular oxidative damage and helps them to maintain endothelial NO signaling