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This fact is justified by the toxic character of the persistent excess of glucose in the organism that ends up promoting glycation of proteins, hyperosmolarity and increase in the levels of sorbitol inside the cells [35]

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Experimental studies carried out in rodents have also shown that bilirubin reduces liver fat accumulation by binding to PPAR (13, 158160), and the mechanism behind this may be an increase in both number and function of mitochondria, leading to a reduction in lipid accumulation (161163)

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This suggests it maintains functional dopamine storage and release mechanisms even when structural components are damaged