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Depletion of peroxiredoxin II promotes keratinocyte apoptosis and alleviates psoriatic skin lesions via the PI3K/AKT/GSK3 signaling axis

In addition to its role in redox buffering, GSH is a regulator of redox-dependent signaling and post-translational modification

Given the pivotal role of PMN-MDSCs in lipid peroxidation-mediated immunosuppression and the resistance of neutrophils/PMN-MDSCs to ferroptosis, these cells pose a significant challenge in combining ferroptosis induction with immune checkpoint inhibition in cancer therapy

The classic mechanism of acetaminophen hepatotoxicity is that its reactive metabolite, N-acetyl-p-benzoquinoneimine (NAPQI), depletes GSH and then covalently binds to proteins

In brief, a suspension of 5 ml (2.5 mg/ml) CB was sonicated in endotoxin-free water for 16 min and used at the same concentration as the i.t

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