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Recent studies have shown that extracellular vesicles (EVs), particularly exosomes (30120 nm vesicles derived from multivesicular body-plasma membrane fusion), as critical mediators of intercellular communication [17, 18]

These conditions could occur due to some other internal and external factors

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This suppression results in reduced production of pro-inflammatory cytokines, such as TNF- and IL-6, which are central to the pathogenesis of fibrosis

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Many previous basic studies support that oxidative stress plays a role in oxaliplatin-related peripheral neuropathy 15,16,17