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Furthermore, it is hypothesized and its expression level is increased that Niacin may function as an anti-atherosclerotic agent by promoting the efflux of lysosomal cholesterol from macrophages via the CD38/NAADP signaling pathway [35]

doi: 10.1146/annurev-immunol-032713-120142 62 TsengDVolkmerJ-PWillinghamSBContreras-TrujilloHFathmanJWFernhoffNBet al