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In addition, IGF-2, whose expression normally is strictly controlled by parental imprinting is upregulated and functions as an important stimulant of the IGF-1R in cancer (LeRoith and Roberts, 2003)

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In accordance with this, TGF- in the TME of MCA-induced fibrosarcoma promoted NK cell conversion into a more immature and less cytotoxic phenotype, defined as ILC1-like, characterized by a pro-angiogenic signature (114)

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[26] Disclaimer: This compounded medication is prepared under section 503A of the U.S

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