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In this regard, it is important to note that COX-2 inhibitors prevent ATL formation during leukocyte-endothelial cell interactions and by inflamed gastrointestinal tract of rats (Fiorucci et al., 2003)

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identified NR1D1 as a potential antimicrobial therapeutic target because it promotes autophagy and lysosomal biogenesis through positive regulation of TFEB expression

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Moreover, the interaction between Bax and Drp-1 has been involved in APAP hepatotoxicity by facilitating mitochondrial fission, which in turn can initiate downstream events, including opening of the MTP

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10.1016/j.cell.2014.06.013 Cell 14 AzzolinL.ZanconatoF.BresolinS.ForcatoM.BassoG.BicciatoS.et al (2012)

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