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A crucial complement component, C5, has become a primary therapeutic target for many inflammatory diseases, including AMD

This oxidative stress will cause the cells to undergo cellular distress and lead to apoptosis, or programmed cell death (under extreme conditions)

This article shows that replicate populations of the same bacterial strain can present different evolutionary pathways in the presence of antibiotics and substantial variations in collateral sensitivity

This process is canonically driven by the stabilization of PTEN-induced kinase 1 (PINK1) on the outer membrane of depolarized mitochondria, which subsequently recruits the E3 ubiquitin ligase Parkin (56)

Braidy N, Grant R, Adams S, Brew BJ, Guillemin GJ (2009) Mechanism for quinolinic acid cytotoxicity in human astrocytes and neurons