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For example, knockdown of PER1 or CRY2 in mammary epithelial cells impairs cell cycle control and facilitates oncogenic behavior, while mouse models with mutations or deletions in circadian genes develop spontaneous tumors or exhibit heightened sensitivity to mammary carcinogens 200

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Notably, the transcription and activity of MBOAT1/2 are subject to modulation by the estrogen receptor (ER) and androgen receptor (AR), which presents a potential avenue for therapeutic intervention in hormone-responsive tumors [30]

Due to the attached acetyl group, the cysteine is protected from oxidation, is released in the liver and is available for glutathione synthesis

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