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Abstract The acid-labile subunit (ALS) of the insulin-like growth factor (IGF) binding protein (IGFBP) complex, encoded in humans by IGFALS , has a vital role in regulating the endocrine transport and bioavailability of IGF-1 and IGF-2

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The possible mechanisms of ALC action in AD may also involve facilitating the rebuilding of cell membranes, as well as improving synaptic function, enhancing cholinergic activity, restoring the brain energy supply, protecting against toxins, and exerting neurotrophic effects via stimulating NGF and the acetylation of proteins [24]

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