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Excess release of cytotoxic ROS during m -Dys leads to OxS, which may hyper-activate platelet function and pose risk of thrombosis in COVID-19 patients

upon exogenous Mn 2+ loading, labile Mn 2+ becomes more broadly distributed, with increased signals observed throughout the cytosol and perinuclear regions in HeLa cells 82

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Accordingly, cells have evolved antioxidant systems to scavenge ROS

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