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TMAO has also been found to promote plaque formation by stabilizing and adjusting the assembly of the amyloid- (A) peptide (Yang et al., 1999) and fibrillation of tau protein by inducing the secondary structure formation at the C terminus in vitro (Scaramozzino et al., 2006)

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We expanded the original questionnaire and weighted the answers, adding sections to evaluate the risk of Lyme and associated co-infections

But backed by a cellular mechanism that addresses one of the biggest blind spots in GLP-1 therapy: what happens to stored fat once appetite drops