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Description

Abstract Background Attention deficit/hyperactivity disorder (ADHD) is a neurodevelopmental disorder characterized by inattentiveness, hyperactivity and impulsivity, which may affect ones cognitive and psychosocial functioning

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In contrast, it has been reported that in the neurons of mice with Fragile X syndrome (FXS), ATP5G1, the c subunit of F 0 -ATP synthase, is abnormally upregulated

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