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Drugs 63 , 845853 (2003)

and it ameliorated the crumpling, intracellular vacuolization and excessive intercellular spacing of pyramidal cells in the hippocampus (Figure 2A)

In that sense, our observations of sustained dysregulated oxidative stress and inflammasome activation in monocytes after short-term recovery support one of the current hypothesis that long term COVID-19 is driven by persistent pathological inflammation and suggest the pathways involved as potential targets for therapeutic intervention (76, 77)