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A fraction of Gln in astrocytes is transported into GABAergic neurons, where it is transformed into Glu (alternatively, Gln in astrocytes is converted into Glu and then transported into GABAergic neurons), which is ultimately converted into GABA by glutamate decarboxylase [4]

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Positron emission tomography studies revealed that lack of sleep also causes inefficient glucose utilization especially in higher-order brain regions such as prefrontal cortex, thalamus, hippocampus and cortex that ultimately lead to impairments in cognitive functions (Wu et al., 1991

Barger et al

Differential regulatory functions of three classes of phosphatidylinositol and phosphoinositide 3-kinases in autophagy