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Furthermore, the current UA elevation therapy shows unstable antioxidant properties, individual variability, and potential adverse effects, thus restricting its clinical applications [14]

For example, Cu deficiency has been shown to promote the aberrant hydrophobicity of mutant SOD1, contributing to the impaired interaction of SOD1 and its neuronal toxicity, and this effect could be reversed by the supplementation of Cu

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And if you're thinking about exact doses and what to take, our glutathione dosage guide has some tips