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Damage to axons is mediated by the following: (1) failure in mitochondrial energy metabolism due to inhibition of the respiratory chain by nitric oxide, which in turn causes a decrease in Na + /K + ATPase activity and alters Na + dependent glutamate transporters, (2) overexpression of glutamate receptors, (3) oligodendroglial excitotoxicity, (4) massive influx of extracellular Ca ++ , (5) activation of proteases, and (6) impaired axonal transport

Lancet 1984;433-5

In almost a third of hypothyroid patients, headache or unilateral head pain is reported as a symptom

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No Phase 1 trial has been conducted, no human pharmacokinetic study has been published, and no incidence percentages for human side effects exist

Treating aplastic anemia is sometimes more difficult