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Tumor radioresistance, in which there is limited cell death following exposure to therapeutic ionizing radiation, is thought to result from several distinct mechanisms, including the proliferation of cancer stem cells (CSCs) leading to tumor regrowth following treatment [17]

Previous studies revealed that, the phosphatidylinositol 3-kinase/Akt (PI3K/Akt) signaling pathway exerts a critical biological role in the cell such as enhancing viability and attenuating ageing, senescence, and death

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[DOI] [PubMed] [Google Scholar] Franklin CC, Krejsa CM, Pierce RH, White CC, Fausto N, Kavanagh TJ

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This was supported by a study by Al-Aubody and Al-Diwan 56 that revealed a higher incidence of liver abnormalities, such as hepatocyte necrosis and central vein congestion, in rats given AAS for 12 weeks