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In healthy astrocytes, nicotinic acid adenine dinucleotide phosphate (NAADP) induces autophagy by activating lysosomal two-pore channels (TPCs), particularly lysosomal calcium channels (TPC2)

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This observation may be due to the distinct nature of the PD depurative treatments, since APD is characterized by shorter and bigger volume stays, which may promote the elimination of carnitine molecules (Di Liberato et al., 2014)

This mechanism effectively decouples NAMPT-mediated NAD + salvage from glycolytic flux, as the resulting low NAD + /NADH ratio thermodynamically inhibits GAPDH

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