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The integration of AI with bioinformatics, de novo protein design, and synthetic biology holds promise for precision therapeutics addressing fungal resistance

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Clinically, alterations in metabolic and redox pathways relevant to ferroptosis, such as reduced glutathione levels and dysregulated cysteine metabolism, have been observed in the brains and plasma of AD patients, providing a human context that anchors the preclinical mechanistic narrative to disease-relevant biology

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