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The other hypothesis suggested that a K + channel protein is the oxygen sensor and inhibition of this channel and the ensuing depolarization is the initial event in transduction, as indicated by Peers and Kemp [1]

Existing studies indicate that in the central nervous system, copper can drive protein misfolding and aggregation to exacerbate neuronal injury, but it can also participate in the activation of antioxidant enzymes and repair factors to inhibit excessive cell death and inflammation, demonstrating a double-edged sword effect

MDA, malonaldehyde

10.3390/md13127060 264 SudP.SuM

Native IGF-1 is rapidly sequestered in circulation by IGF-binding proteins (IGFBPs 16), which extend its half-life but also restrict bioavailability