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[DOI] [PMC free article] [PubMed] [Google Scholar] 157.Campbell N.K., Fitzgerald H.K., Dunne A

These findings confirmed that the role of CTPS1 in DNA damage repair is primarily dependent on H1, and that H1(N76D/N77D) is the functional modification site that regulates CTPS1-mediated DNA damage repair

Also, this disturbed BCAAs metabolism has a synergistic effect with the development of T2DM

In elderly individuals administered a single 2000 mg UA capsule, the majority of UA detected in skeletal muscle was in its monomeric form, with only a few subjects showing trace amounts of UA-glucuronide (7)

References Franceschi, C

320 QKI is suppressed in a variety of tumors, and its expression levels are strongly correlated with the aggressiveness, metastatic potential, and clinical outcomes of these tumors