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It appears to work in part by promoting angiogenesis, the formation of new blood vessels, which accelerates delivery of repair signals to damaged tissue

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To recover the mitochondrial functions and to reduce oxidative stress, inhibition of CypD activation using cyclosporin A (Duann and Lin, 2017) and N-methyl-4-isoleucine cyclosporine (Niimi et al., 2014) has been reported