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Host antiviral immunity requires an optimal and coordinated response to control viral infections

When the lungs are stimulated by infection or oxidative stress, immune cells (e.g., alveolar macrophages and dendritic cells) release inflammatory cytokines such as TNF- and IL-6 [442], which enter the brain via the bloodstream

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A case control study examining this issue found no increased risk of this condition except in patients with a history of MI

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The extent and predominance of type 2, type 3 or a lack of overt inflammation among patients with different fibrotic diseases is an open area of research requiring larger patient cohorts of diverse genetic backgrounds