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A cystine-glutamate antiporter then exchanges this cystine for glutamate, activating inhibitory metabotropic glutamate receptors named Group II metabotropic glutamate receptors (mGlu2/3 receptors) on glutamatergic nerve terminals and lowering synaptic glutamate release

This finding is in agreement with Anitua et al., who reported that endogenous activation supports a sustained release of growth factors, in contrast to exogenous activation, which induces a rapid initial burst followed by early depletion 38

Revealing the dynamic mechanism by which transferrin promotes the cellular uptake of HAIYPRH peptide-conjugated nanostructures by force tracing

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The high-affinity NMDA-Rs on the beta cell are already saturated at physiological glutamate concentrations in the islet, and are mainly activated through depolarization of beta cells [121]