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We establish that in TNBC tumors from obese mice, GPX4-mediated redox defense is a targetable vulnerability, and that GPX4 is a powerful regulator of treatment response in our metM-Wnt lung model of TNBC and obesity

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Receptor-mediated endocytosis is also known as clathrin-dependent endocytosis as the membrane-associated protein clathrin takes part in forming membrane vesicles that become internalized into the cell (32)

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