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There are also molecular pathways that can function as upstream mediators of TGF- in pathological events

To dissect the biochemical derangements imposed by chronic hyperglycaemia, a three-dimensional, tissue-engineered wound model has been engineered that faithfully recapitulates the accumulation of AGEs and the attendant impairment of fibroblast proliferation, angiogenic signaling, and extracellular-matrix turnover that underlie the stalled diabetic repair program (129)

The citation is: Kim, T., Deng, Y., Hwang, Y., Hernandez, L., Patel, M., Kirk, N., Berg, A., Yli-Piipari, S

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The loss of astrocytes reduces the density of oligodendrocytes, initiates demyelination, and promotes myelin abnormalities (e.g., decompaction) in the white matter of developing and adolescent mice (7 days to ~1.5-months-old

doi: 10.1128/iai.72.8.4410-4415.2004 CrossRef Google Scholar View reference in article 21 ChienJYHsuehPRChengWCYuCJYangPC