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291 OksanenM.LehtonenS.JaronenM.GoldsteinsG.HmlinenR.KoistinahoJ

Diallyl disulfide disrupts the tumor cell cycle by activating the protein ERK

It is the source of numerous bioactive lipid second messengers in inflammation and oxidative stress

This molecular mechanism can be attributed to the spongelike activity of UCA1, as it sequesters miR16 and blocks it from attaching to the GLS2 mRNA's 3'UTR region, thereby inhibiting the degradation of GLS2 mRNA (Figure 2F) [189]