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Engineering the enantioselectivity of glutathione transferase by combined active-site mutations and chemical modifications Biochim

The potential of using circulating tumour cells and their gene expression to predict docetaxel response in metastatic prostate cancer

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and (ii) off-target resistance caused by the activation of bypass signal transduction resulting from the amplification of the ERBB family of receptor tyrosine kinase genes, BRAF amplification, KRAS amplification, and KRAS mutations [148]

Ruxolitinib as salvage therapy for chronic graft-versus-host disease

Attempts at withdrawal are more likely to succeed if the patient is able to contemplate the ultimate goal of cessation, and the doctor and patient are able to work together in a productive therapeutic relationship to achieve this