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Loss of farnesoid X receptor (FXR) exacerbated the ferroptosis signaling pathway by enhancing iron accumulation, increasing lipid peroxidation, decreasing GSH levels and reducing GPX4 expression in cisplatin-treated mice and HK-2 cells [175]

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In preclinical research models, exposure to GHK-Cu has been associated with altered gene expression patterns and modulation of signalling pathways related to extracellular matrix dynamics, oxidative stress response, and cellular turnover