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For the AMP treatment group, significant enrichment was observed in pathways related to riboflavin metabolism, phenylalanine metabolism, and the biosynthesis of phenylalanine, tyrosine, and tryptophan (Figure 3B)

Moreover, PLBP mitigated neuroinflammation and offered indirect protection to RGCs by inhibiting microglial phagocytosis and migration and the release of proinflammatory cytokines and by activating the NF-B pathway

An ideal therapeutic approach should aim to inhibit pyroptosis or necroptosis during the acute inflammatory phase, while restoring apoptosis or autophagy during the resolution phase to eliminate pathogenic cells and promote tissue repair

Palmitoylation-dependent estrogen receptor alpha membrane localization: regulation by 17beta-estradiol

Acknowledgements We would like to thank all volunteers for their participation in this study and Drs Tomas Jelenik, Chrysi Koliaki, Daniel Markgraf for their important contributions to the clinical examination and/or laboratory assessments as well as Kai Tinnes, Myrko Esser, David Hhn, Andrea Sparla, Kerstin Frster, Fariba Zivehe, Jan-Marc Leonhard, Michelle Reina Do Fundo, Karin Rhrig and Ulrike Partke for their excellent technical assistance

doi:10.1016/j.diabres.2024.111717 Rodriguez PJ, Goodwin Cartwright BM, Gratzl S, et al