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Investigations into the possible mechanism showed that allicin inhibited the apoptosis of astrocytes induced by hypoxia via GPX1, activating SRC-AKT-ERK and lipid peroxidation

Regio- and enantioselective hydrolysis of epoxide c

It is capable of combating oxidative stress within the body, which can result in cell damage and is associated with a multitude of chronic conditions [1]

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Perhaps the redox-signaling roles of the TrxR1 system are so crucial in metazoans that its ancestral redox-homeostasis roles must be excused, when necessary, for the sake of sustained effective signaling