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qIodine status influences apoptosis: high iodine intake can induce Fas-dependent thyrocyte apoptosis, whereas restriction attenuates it(Pyzik et al

Potentiation of lead-induced cell death in PC12 cells by glutamate: Protection by N-acetylcysteine amide (NACA), a novel thiol antioxidant

Inhibition of GSH metabolism leads to a decrease in myeloid-derived suppressor cells (MDSCs) and facilitates their conversion into M1 macrophages, which promote the process of cross-presentation for tumor antigens and activate CD8 + T cells, thereby performing anti-tumor functions [14]

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1 A ) could shape excitatory transmission, we measured the frequency of miniature excitatory postsynaptic currents (mEPSCs), a reflection of presynaptic drive