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As evidenced by a similar negative slope in their respective regression lines, neither of the anticoagulants perfectly prevent spontaneous oxidation of free GSH

10,14 The regulation of GCL expression and activity is critical for the homeostasis of GSH

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In addition to biofilm disruption, our study demonstrated that GSH facilitates epithelial cell re-growth and confluence by restoring intracellular GSH levels and enzymatic activity

Astragaloside IV and cycloastragenol are equally effective in inhibition of endoplasmic reticulum stress-associated TXNIP/NLRP3 inflammasome activation in the endothelium

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