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Acylcarnitine is released from abnormal mitochondria in peripheral glial cells to affect surrounding axons, resulting in the destruction of axonal membrane properties and the induction of axonal degeneration, which has been shown to be neurotoxic [116]

These DSBs are usually repaired by homologous directed repair (HDR) or nonhomologous end joining (NHEJ), achieving mutation or foreign gene insertion (Fig

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