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Furthermore, OHP causes damage to both neuronal and nonneuronal mitochondria, leading to oxidative stress burden mediated by the redox-sensitive TRPA1 channels, which correlate with OHP-induced cold and mechanical hypersensitivity [71, 115,116,117]

Multiple animal and cell-culture studies have shown that when renal glutathione is experimentally depleted, tubular cells become significantly more vulnerable to injury from toxins, ischemia, and oxidative stress reinforcing that adequate glutathione is protective for kidney tissue, not threatening to it

a resident pool of immune cells (for example, macrophages and synovial fibroblasts) is stimulated

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10.1097/01.hjh.0000500039.08261.4f 142 WanF.MaF.WuJ.QiaoX.ChenM.LiW.et al (2022)

Many trichome development studies have focused on model plants such as Arabidopsis