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(89) found that Guizhi Fuling Wan significantly reduces concentrations of inflammatory factors like interleukin-6 (IL-6), tumor necrosis factor- (TNF-), and high-sensitivity C-reactive protein

In addition, PYC suppresses adipose differentiation-related protein, which is involved in atherosclerosis development and highly expressed in macrophages ( In an in vitro study on microglial cells, PYC dose-dependently decreased the release of nitric oxide, tumor necrosis factor- (TNF-), interleukin (IL)-6, IL-1, ICAM-1, and perilipin 2, thereby exhibiting anti-inflammatory activity ( Ex vivo analysis of plasma from volunteers following PYC administration revealed moderate inhibition of COX-1 and COX-2 activity, indicating potential anti-inflammatory and platelet-inhibitory effects (Schfer et al., 2006)

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These models offer powerful tools for identifying latent disease subtypes, modeling progression trajectories, and predicting therapeutic responses, capabilities that are crucial for personalizing neuroprotective strategies [523, 590592]

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