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Glutathione depletion activates mitogen-activated protein kinase (MAPK) pathways that display organ-specific responses and brain protection in mice

515 The pro-apoptotic molecule CHOP underscores the connection between ER stress and cell fate in MS, with its expression in various cell types suggesting immune-mediated processes contributing to ER stress-associated damage

Metabolic reprogramming and immune escape are considered to be the main characteristics of malignant tumors, which can be used to diagnose, detect and treat tumors [1, 2].In the past few decades, the Warburg effect has been considered as a typical example of metabolic reprogramming, wherein the tumor cells still rely on the conversion of glucose to lactate for energy needs under aerobic conditions, which is crucial for tumor initiation and progression [3, 4]

6, severe injury (up to 75% injury of the field of view)

Role of p53 in the regulation of cellular senescence

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