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GPX4 aggravates experimental autoimmune encephalomyelitis by inhibiting the functions of CD4 T cells

Mechanistically, several MGBA pathways appear to be involved in AD

In fact, the high expression of glycogen synthase-1 in RA-FLSs can lead to the excessive accumulation of glycogen and inhibit AMPK expression, leading to the high expression of matrix metallopeptidase (MMP)-1, MMP-9, IL-6, and CCL-2, along with increased proliferation and migration of FLSs

MuddSHBrosnanJTBrosnanMEJacobsRLStablerSPAllenRHet alMethyl balance and transmethylation fluxes in humans

[DOI] [PubMed] [Google Scholar] 19.Sreedharan J., Blair I

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