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Thus, N-acetyl-L-cysteine lowered blood pressure in hypertension (Tang et al., 1991), restored the content of GSH during myocardial infarction and heart failure in mice (Adamy et al., 2007)

In mice, CHAC1/BOTCH promotes neuronal differentiation by inhibiting NOTCH1 and both its depletion and overproduction severely disrupt neurogenesis ( CHAC1/BOTCH in zebrafish leads to severe defects in brain, heart and myotome development, resulting in embryonic lethality, highlighting its conserved and essential role (Yadav et al., 2019)

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[3] To avoid mobilization, some practitioners of chelation use strong chelators, such as selenium, taken at low doses over a long period of time

Significant neurologic and motor deficits may occur following ischemic stroke