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The results indicate that the apoptosis rate increases as the concentration of quercetin increases ( Furthermore, quercetin induces endoplasmic reticulum (ER) stress by increasing UPR-related gene expression and promoting PERK and IRE1 phosphorylation, thereby activating apoptosis in hepatic stellate cells ( 3.2.2 Quercetins engagement with autophagy pathways Autophagy, a critical cellular mechanism for the degradation and recycling of cytoplasmic components, plays a pivotal role in maintaining cellular equilibrium, adapting to stress, and ensuring cellular vitality (Takeshige K et al., 1992)

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