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We found that 24 h or 48 h treatment with GYY4137 at varying concentrations shows no significant differences in cell viability compared with the vehicle-treated group, except for the 400 M/48 h treatment group (Fig

The dual nature of type I and type II interferons

The increase in ROS can also affect the apoptosis and metabolism of glioma cells via the JNK-regulated metabolic pathway and ataxia telangiectasia mutated (ATM)-Yes-associated protein 1 (YAP1)-driven apoptotic pathway ( Celastrol is one of the most important active ingredients of the traditional Chinese medicine Tripterygium wilfordii , which activates the JNK pathway and ROS production and inhibits the activities of mechanistic targets of rapamycin (mTOR) and Akt kinases, significantly increasing apoptosis and autophagy in glioma cells (Liu et al., 2019)

M., Humadi, A

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Ddr2 Upregulation Confers Ferroptosis Susceptibility of Recurrent Breast Tumors through the Hippo Pathway