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The rate limiting amino acid in de novo synthesis of GSH in metabolic disease appears to be glycine

Moreover, the primary function of glutathione peroxidase enzymes is to protect cells from oxidative damage by reducing peroxides and preventing their harmful effects

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Consistently, in the GLS-deficient mouse tumor model, the increased concentration of glutamine in the TME due to restricted glutamine utilization by tumor cells leads to elevated levels of glutamine available to tumor-infiltrating T lymphocytes, thereby enhancing its anti-tumor activity [17]

For optimal formation, recycling, and synergistic supportat the cellular level