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Several studies identified DNA-sensing pattern recognition receptors (PRRs) as drivers of IFN I-driven inflammation and massive cell death in severe COVID-19: cyclic GMPAMP synthase (cGAS), absent in melanoma (AIM)2, nucleotide-binding oligomerization domain (NOD)-like leucine-rich repeat (LRR) and PYRIN domain containing (NLRP)3, receptor for advanced glycation end products (RAGE), and Toll-like receptor (TLR)9 (9, 13, 14, 1723): Upon detecting double stranded (ds)DNA, these PRRs activate the production of antiviral and pro-inflammatory cytokines and chemokines and eventually, cell death, via two principal pathways (reviewed in (24, 25))

Mutations in LRRK2 linked to parkinson disease sequester Rab8a to damaged lysosomes and regulate transferrin-mediated iron uptake in microglia

doi: 10.3390/nu12092785 135 RakhraVGalappaththySLBulchandaniSCabandugamaPK

10.1016/S0140-6736(23)01419-8 23 BertouxM.CassagnaudP.LebouvierT.LebertF.SarazinM.Le BerI.et al (2020)