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3d), while the AMPAR antagonist CNQX (which prevents BiC/4-AP-induced bursting that leads to NMDAR activation) did inhibit protection (Supplementary Fig

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Collectively, our data suggest that the PARG inhibitor PDD promotes targeted degradation of BRD2/3/4 via enhanced BRDPROTACCRL2 VHL ternary complex formation and K29/K48-linked ubiquitylation

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Spontaneously formed methemoglobin is normally reduced (regenerating normal hemoglobin) by protective enzyme systems, e.g., NADH methemoglobin reductase (cytochrome-b5 reductase) (major pathway), NADPH methemoglobin reductase (minor pathway) and to a lesser extent the ascorbic acid and glutathione enzyme systems

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