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Contribution of Bcl-2, but not Bcl-xL and Bax, to antiapoptotic actions of hepatocyte growth factor in hypoxia-conditioned human endothelial cells

It has been further suggested that the proteolytic cleavage of HGF could trigger its structural rearrangement that is likely to be critical for inducing c-MET activation 11,12

These newly formed capillaries deliver oxygen, nutrients, and growth factors directly to injured tissue