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Further study showed that Tim-AIII promoted HSP90 expression and Tim-AIII induced cell death was blocked by the HSP90 inhibitor tanespimycin, indicating that HSP90 was the main target of Tim-AIII to further trigger intracellular events

Identifying these mutations may guide preventive measures and treatment

Methods Clinicopathological data were collected from 460 breast cancer patients who underwent adjuvant chemotherapy from January 2016 to December 2019 from Zhengzhou University Peoples Hospital

2015 Aug 18;314(7):731

Borchert A, Kalms J, Roth SR et al (2018) Crystal structure and functional characterization of selenocysteine-containing glutathione peroxidase 4 suggests an alternative mechanism of peroxide reduction

Importantly, this early cataract formation is closely associated with alpha A-crystallin glutathionylation accumulation and compromised mitochondrial functions in gene KO mice [37]