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In breast cancer cells, a high level of miR-378* induces the metabolic shift from an oxidative to a glycolytic bioenergetics pathway by inhibiting the expression of two PGC-1 partners, ERR (estrogen-related receptor gamma) and GABPA (GA binding protein transcription factor, alpha subunit)

In addition to the rapid and long-lasting antidepressant response, boosting mitochondrial metabolism of LAC leads to the amelioration of specific cognitive domains [46, 47], and promotes behavioral resilience at the social defeat stress (SDS) paradigm [27]

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Recent developments in myofibroblast biology: paradigms for connective tissue remodeling